Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein with Antibodies | ACS Chemical Biology
Frontiers | Key Metabolic Pathways in MSC-Mediated Immunomodulation: Implications for the Prophylaxis and Treatment of Graft Versus Host Disease
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Frontiers | Regulation of Age-Related Protein Toxicity
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Frontiers | Squalamine and Its Derivatives Modulate the Aggregation of Amyloid-β and α-Synuclein and Suppress the Toxicity of Their Oligomers
Alliance for Healthy Aging
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Julius Bier Kirkegaard — Theoretical Biophysics & Data Science
The therapeutic potential of targeting tryptophan catabolism in cancer | British Journal of Cancer
Speakers | ARDD 2022
A Brain-Permeable Aminosterol Regulates Cell Membranes to Mitigate the Toxicity of Diverse Pore-Forming Agents | ACS Chemical Neuroscience
Frontiers | Parasexuality of Candida Species
Cornelis CALKHOVEN | University of Groningen, Groningen | RUG | European Research Institute for the Biology of Ageing | Research profile
Frontiers | Cellular Regulation of Amyloid Formation in Aging and Disease
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Delivery of Native Proteins into C. elegans Using a Transduction Protocol Based on Lipid Vesicles | Scientific Reports
Untargeted Metabolomic Characterization of Glioblastoma Intra-Tumor Heterogeneity Using OrbiSIMS | Analytical Chemistry
Cognitive impairments correlate with increased central nervous system immune activation after allogeneic haematopoietic stem cell transplantation | Leukemia
Proteome-wide Changes in Protein Turnover Rates in C. elegans Models of Longevity and Age-Related Disease - ScienceDirect
Akira KITAMURA | Senior Lecturer | Ph.D. | Hokkaido University, Sapporo | Hokudai | Faculty of Advanced Life Science | Research profile
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect
Frontiers | Regulation of Age-Related Protein Toxicity
Deletion of SERF2 in mice delays embryonic development and alters amyloid deposit structure in the brain | Life Science Alliance
